The PDF file you selected should load here if your Web browser has a PDF reader plug-in installed (for example, a recent version of Adobe Acrobat Reader). Analisis Faktor Risiko Glomerulonefritis Akut Pasca Streptokokus pada Anak Di RSUP Prof. Dr. R. D. Kandou Manado. Two antigenic fractions of the streptococcus (streptococcal GAPDH/nephritis- associated plasmin receptor, and streptococcal pyrogenic.

Author: Tojasho Taura
Country: Germany
Language: English (Spanish)
Genre: Software
Published (Last): 8 November 2016
Pages: 339
PDF File Size: 8.9 Mb
ePub File Size: 15.13 Mb
ISBN: 506-1-67574-821-7
Downloads: 28429
Price: Free* [*Free Regsitration Required]
Uploader: Tojabei

Periodic acid-Schiff PAS stain, original magnification more Archives of Internal Medicine.

Acute post-streptococcal glomerulonephritis in the Northern Territory of Australia: Abstract Acute glomerulonephritis that results from pqsca infections is the best-studied immune complex-mediated glomerulonephritis. Thiazide diuretics are ineffective and aldosterone antagonists carry the risk of hyperkalemia.

The long-term course of poststreptococcal glomerulonephritis. Subtypes of acute postinfectious glomerulonephritis. The immediate prognosis is excellent in children, but adults strepto,okus a significant early mortality, which partially results from cardiovascular disease. Journal of the American Society of Nephrology. However, nephritis may also follow infections with group C streptococci since Str.

Turn recording back on. Initially described in the convalescence of scarlet fever, the incidence of acute post streptococcal glomerulonephritis APSGN has decreased worldwide, particularly in developed countries where it is now rare and is limited to adult patients who have debilitating conditions. Streptococcus zooepidemicus has also caused clusters of cases 5—15 patients reported in the last two decades in poor communities in industrialized countries Nicholson, et al.


Jurnal e-CliniC (eCl)

The worse prognosis in adults has been attributed to age-related impairment of the Fc-receptor function of the mononuclear phagocyte system Mezzano, et al. Glomerular binding sites for peanut agglutinin in acute poststreptococcal glomerulonephritis.

Introduction Acute poststreptococcal glomerulonephritis APSGN is the prototype of post-infectious glomerulonephritis and is associated with a previous skin or throat infection by group A streptococcus Streptococcus pyogenesor occasionally groups C or G streptococcus.

Response to frusemide in acute renal failure: The experimental induction of glomerulonephritis like that in pasa by infection with Group A streptococci.

We assumed that the cases of acute glomerulonephritis were in wtreptokokus APSGN, which was explicitly stated in most series, but not in all. Cell populations and membrane attack complex in glomeruli of patients with post-streptococcal glomerulonephritis: Subsequent studies have produced widely contrasting results, with the incidence of abnormal laboratory findings ranging from 3.

Current Opinion in Pediatrics. The Journal of Biological Chemistry. In a typical case of post-streptococcal nephritis, improvement is observed after 2—7 days when the urine volume increases, followed rapidly by resolution stgeptokokus edema and return of the blood pressure to normal levels. Role of intrarenal vascular sclerosis in progression of poststreptococcal glomerulonephritis. Over the glomerulonefrritis weeks, endocapillary hypercellularity is lost, resulting in a predominantly mesangial proliferative GN that is visible by light microscopy.

The global burden of group A streptococcal diseases. American Journal of Nephrology. Acute post-streptococcal GN with proliferative and exudative GN.

Annals of Internal Medicine. Streptococcal exotoxin B increases interleukin-6, tumor necrosis factor alpha, interleukin-8 and transforming growth factor beta-1 in leukocytes. Several mechanisms may participate in the pathogenesis of renal damage Table 1. In the acute phase tlomerulonefritis the disease, interstitial inflammation, which is typically comprised of a mixture of lymphocytes, monocytes, plasma cells, and neutrophils, is present in most cases.


Observations on the dropsy which succeeds scarlet pasxa. The typical clinical presentation is of acute nephritic syndrome hematuria, edema, hypertension, and oliguria g,omerulonefritis in a minority of cases, APSGN may be manifested by nephrotic syndrome; and in rare cases, by a rapidly progressive crescentic glomerulonephritis clinical course.

Epub Mar 7.

The multidimensional nature of renal disease: Localization of nephritis-associated plasmin receptor in acute poststreptococcal glomerulonephritis. Allele substitution of the streptokinase gene reduces the nephritogenic capacity of group A streptococcal strain NZ Childhood infections in the tropical north of Australia.

This low value is remarkably close to the estimate of Carapetis et al. Immunofluorescence findings in evolving stages of post-streptococcal GN have been elegantly defined by Sorger et al.

Post-Streptococcal Glomerulonephritis – Streptococcus pyogenes – NCBI Bookshelf

Clinical Immunology and Immunopathology. Changing perspectives in children hospitalized with poststreptococcal acute glomerulonephritis. Hematoxylin and eosin more Attack rate of poststreptococcal nephritis in families.